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Sabina, R

503,512,518 These effects include modulating cell proliferation, inflammation, and oxidative stress in liver cells, all of which are key factors in the development and progression of HCC

Rodents overexpressing USP22 or lncRNA PART1 were well resistant to I/R-induced oxidative stress and cardiac injury, which may be partly attributed to the elevated GSH content in the myocardium [251, 252]

This review synthesizes current evidence on the molecular and clinical mechanisms of atorvastatin-induced hepatotoxicity, emphasizing genetic susceptibility, transporter interactions, and mitochondrial dysfunction

Recent Findings SGLT2-i consistently lower office SBP/DBP (2.54.0/1.52.0 mmHg) and 24-hour ambulatory BP (3.8/1.8 mmHg)