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The development of thyroid autoimmunity requires initial interactions between thyroid follicular cells, antigen-presenting cells (APCs), and T lymphocytes

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These metabolites, including acetic acid, lactic acid, formic acid, and other SCFAs, reduce intracellular pH, creating an acidic microenvironment that is hostile to pathogen growth and proliferation (196)

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Peer-reviewed literature indexed on PubMed Reviews GHK-Cu modulation of gene expression related to tissue remodeling and anti-inflammatory pathways