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ERK1/2 activation triggers transcription factors c-Fos and c-Jun that coordinate cellular repair responses
However, a more recent study observed that physiological levels of oncogene (KRAS G12D and MYC) expression could trigger a NRF2-dependent antioxidant response to downregulate ROS levels in cells [8]
Option 3: The Middle Ground Increasingly, employers are choosing a balanced approach
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