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Returning to our experiment with gilts and the profiles of Se-GSH-Px (Figures 1 and 2), the selenium status of the control animals was inadequate and Se GSH-Px declined, which is in agreement with an oxidative pressure brought by the peri-estral period
Meta-analysis was performed using a random-effects model
The results showed that there was a dose-dependent migration of HUVECs toward CCL11, indicating that CCL11 released by THP-1 cells can affect endothelial cell function by its chemotaxin essence (Fig
Lee BJ, Lin JS, Lin YC, Lin PT, BioMed Central Ltd
2e,f), making the existence of similar substrateenzyme interactions in other arGSTFs plausible
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