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TMAO can aggravate acute electrical remodeling in AF models and enhance atrial electrophysiological instability in normal canines by enhancing autonomic remodeling and boosting the p65 NF-B signaling pathway [84]

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This, in turn, doubles the capacity of the available growth factors to promote signaling cascades necessary for mitogenesis (cell division), motogenesis (promotion of cellular motility), morphogenesis (structural development), neurogenesis (growth and development of nervous tissue), production of stem cells, and protection of a wide range of cells against injury