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Although the knockout of GPX7 does not lead to the direct death of mice, the production of excess reactive oxygen species leads to incorrect oxidative modification of proteins and irreversible changes in function, resulting in various diseases
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In in vitro experiments, NRF2 expression was reduced in IL-22-stimulated HaCaT cells, while the addition of SFN partially restored NRF2 expression
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Kent Holtorf, a physician who graduated from UCLA School of Medicine
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