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Recent Findings Preliminary data suggest that GLP-1 RA can modulate crucial pathways in the pathogenesis of IBD, such as chronic inflammation circuits, intestinal tight junctions, and gut microbiome dysbiosis, setting the stage for human trials to investigate the role of these agents in the treatment of IBD among people with or without diabetes and obesity

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Notably, ABCG2 assumes a prominent role in urate excretion within the intestines, surpassing its involvement in other tissues, thus emerging as the primary conduit for extra-renal urate elimination [12, 52]

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