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Here, working primarily in mouse hepatocytes, we show that decreasing the availability of substrate for the TCA cycle diminished NADPH production and attenuated ER stress in a manner that depended on glutathione oxidation

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Strikingly, this form of hepatotoxicity is also associated with cerebral abnormalities such as anxiety, disorientation, sluggish speech, and even loss of consciousness and coma that typically manifest in the form of hepatic encephalopathy 13,14,15,16,17

Seek urgent medical attention if you develop sudden or severe neurological symptoms, such as significant weakness, loss of coordination, visual disturbances, or confusion, as these may indicate more serious neurological complications requiring immediate assessment