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"These findings have immediate and potentially transformative implications for how these blockbuster drugs are prescribed and for the development of new, more effective treatments with better tolerability," noted Mayo Clinic researchers in the paper titled "A GENETIC RISK SCORE ASSOCIATED WITH NAUSEA RESULTING FROM GLP-1 AGONIST TREATMENT." Additional findings from the study revealed: High-CTSGRS patients were also significantly more likely to report headaches (odds ratio of 5.4) A trend indicating individuals with high-CTSGRS were more prone to experiencing GI-related side effects in general (73% vs

GLP-1-activated PKA inhibits voltage-gated potassium channels, prevents membrane repolarization, and prolongs VDC channel opening to promote Ca 2+ inward currents, contributing to GLP-1 stimulate insulin secretion, and the antagonistic effect of GLP-1R activation on Kv currents in cells requires transactivation of epidermal growth factor receptors, accompanied by signal conduction of cAMP/PKA and phosphatidylinositol-3-kinase (PI3K)/PKC signaling (MacDonald et al., 2003)

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