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Another possibility is that inhibition of proteasome system may affect Ku80 turnover at damages sites, thus impairing its removal and consequently NHEJ repair activity (Feng and Chen, Based on our results, we cannot state that Ku80 exclusively accounts for NHEJ dysfunction observed in HSV-1 infected neurons
In vivo models of primary brain tumors: Pitfalls and perspectives
However, AKI-mediated effects in the liver have not been comprehensively assessed
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