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It was released as a blood glucose control treatment for patients with diabetes, but it was approved for use as an obesity treatment by the US Food and Drug Administration (FDA) in 2014, as it was found to be effective for weight loss without hypoglycemia in obese people who did not have diabetes
Taken together, these findings highlight the common mechanism of action of GLP-1R in DM and AD and its potential to regulate metabolism and neuroprotection (Figure 2), suggesting that GLP-1R may be a key factor linking these two diseases
Some clients see improvements with regular sessions
Peptides 2018;100:4853.ArticlePubMed 108
It has been demonstrated that GSH depleted astrocytes lose their neuroprotective roles, leading to neuronal toxicity and a consequent reduction in cell viability (Pizzurro et al., 2014a,b)
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