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Dual checkpoint blockade of CD47 and LILRB1 enhances CD20 antibody-dependent phagocytosis of lymphoma cells by macrophages

Escape latency was recorded, and the mean of the four starting points was used for data analysis

That is the mechanistic case for the blend in one paragraph and it is exactly why the two are described as complementary rather than redundant

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BPC-157s modulation of pro-inflammatory cytokines, without suppressing normal immune function the way corticosteroids do, makes it a useful tool for patients who need repeated joint management but want to avoid the tissue-degrading effects of repeated cortisone injections