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2.1 L-arginine metabolism Leishmania is critically dependent on L-arginine for survival ( Leishmania also expresses key enzymes involved in L-arginine metabolism, including arginase (ARG), and ornithine decarboxylase (ODC) ( Given its importance for parasite survival, the L-arginine metabolic pathway represents a key target of host defense ( -hydroxy-l-arginine, leads to a decrease in parasite growth ( Leishmania- infected macrophages with arginase inhibitor decreases intracellular parasite burden by suppressing both host and parasite arginase activity ( -difluoro-methyl ornithine (DFMO), and 3-aminooxy-1-aminopropane (APA), restrict parasitic infection ( Leishmania has evolved its L-arginine metabolic machinery to compete with the host for this essential nutrient in order to survive and proliferate ( 2.2 Polyamine pathway Polyamines (PA), including putrescine, spermidine (Spd), and spermine, are produced via the ARG pathway, which is induced in alternatively activated and metabolically suboptimal macrophages ( Leishmania from peroxynitrite-mediated cytotoxicity ( Several PA biosynthetic enzymes are essential for Leishmania survival and represent promising novel drug targets

By bypassing the digestive system, these infusions provide essentially 100% bioavailability for essential nutrients that support many different autoimmune conditions

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[PMID: 10503467] PubMed record related to this compound for laboratory literature review