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This was further substantiated by the observation that inhibiting heightened GJ function with a GJ inhibitor in mice effectively diminished APAP toxicity, which correlated with reduced levels of oxidative stress in hepatocytes
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10.3390/v12091008 61 SuL.QuH.CaoY.ZhuJ.ZhangS
Its chemical synthesis was reported long back in 1929 and is a derivative of the naturally occurring selenoaminoacid, selenocystine [80, 81]
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