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While these results cannot be directly extrapolated to human pulmonary hypertension patients, they suggest BPC-157 may influence the vascular remodeling processes that underlie this condition

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and 3-numerous confluent changes subcategorized into: (a) the hemorrhagic zone, (b) edema, (c) the loss of neurons, (d) vacuoles, and (e) the loss of spinal column tracts [3]