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It was therefore concluded that NiCl 2 induces p38 MAPK-dependent lytic cell death and stimulates markers of premature eryptosis, which may explain the changes in hematological parameters observed in poisoning victims [420]
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The principle of the EPR effect is based on the rapid and uncontrolled growth of cancer cells, which enables them to reprogram their vasculature to replenish the increasing demands of oxygen and nutrients, through the formation of structurally abnormal tumor capillaries and blood vessels having defects in their basement membrane with discontinuous epithelium [86, 87]
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