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Evidence shows that the strongest genetic risk factor for late-onset AD is the E4 allele of the cholesterol transporter APOE (APOE4), besides, APOE4 could promote amyloid aggregation and impair clearance from the brain directly binding to A (63)
Binding of glutathione and an inhibitor to microsomal glutathione transferase
It was also evaluated in Phase 2 clinical trials by Helsinn Therapeutics for postoperative ileus, though that programme was discontinued
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