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By mimicking the natural incretin hormone GLP-1, these medications activate specific receptors throughout the body, triggering glucose-dependent insulin secretion, suppressing glucagon release, slowing gastric emptying, and modulating appetite centres in the brain
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J.et al (2014)
Furthermore, it slightly reduced the increase of glucose transporter types 1 and 4 (GLUT1/4) during adipogenesis and led to a rise of GLP-1 mRNA, though protein levels remained unchanged
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