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An alternative strategy of starting the diagnostic investigation with mRNA sequencing has also been recommended, particularly because of the delay in diagnosis that can occur when glutathione synthetase deficiency results from a splice site mutation (02)

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Although we determined that a wide range of Tie2 + ECs and HL cells are not required for the antiatherogenic actions of semaglutide, our studies did not identify the precise GLP-1R + cells required for GLP-1RAs to attenuate atherosclerosis

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