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It was identified in the late 1980s and initially attracted attention because its deposits are found in the pancreases of people with type 2 diabetes

It stimulates lipolysis (the breakdown of stored fat) and inhibits lipogenesis (the formation of new fat)

In a retrospective cross-sectional study of 165 patients with thromboembolic and other cardiovascular manifestations identified among 1006 hospitalized patients, 165 patients (84%) had at least moderate hyperhomocysteinemia with levels greater than 30 mol/L, 27% had levels greater than 50 mol/L, and 3% had severe elevations greater than 100 mol/L (148)