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doi: 10.3389/fcvm.2018.00068 56 Da DaltLCabodevillaAGGoldbergIJNorataGD

The reduction in VLDL appears to be driven not only by reduced hepatic output but also by accelerated catabolism of apoB 100 containing lipoproteins via upregulation of LPL (lipoprotein lipase) activity and downregulation of apoCIIIthe same pathway implicated in postprandial TRL clearance above

The potential antidepressant effects of exogenous ALC in alleviating depressive-like behavior may be attributed to the modulation of NO and, consequently, the modulation of serotonin and other neurotransmitters, as well as attenuating oxidative/nitrosative stress, affecting NO production in the PFC

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