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Abstract Background Heart failure (HF) is a major cardiovascular complication of type 2 diabetes mellitus (T2DM)
Thus, genome-scale metabolic models of host cells extended to incorporate viral replication 16 can be employed to identify host enzymes essential for viral replication but dispensable for cellular viability
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Trypanosoma cruzi infection disturbs mitochondrial membrane potential and ROS production rate in cardiomyocytes
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