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In mouse models of DKD and TGF-treated HK-2 cells, pharmacological inhibition of ferroptosis via the selective inhibitor Ferrostatin-1 significantly suppresses HK-2 ferroptosis, attenuates tubular injury and renal damage (81)

Without heavy metals, knockout or overexpression of gr did not affect the growth curves on S 0 compared with the control strains (Figure 7A), suggesting that GR did not play a key role in the growth on S 0

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