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A 2016 stability study on GH-releasing peptides showed measurable potency loss after 72 hours of exposure to standard fluorescent laboratory lighting at room temperature [8]
But a person on semaglutide is experiencing GLP-1 receptor activation at levels that dwarf anything your body naturally produces, because synthetic GLP-1 receptor agonists are designed to be resistant to the DPP-4 enzyme that rapidly degrades natural GLP-1
The theoretical rationale for combination is sound, but no controlled human study has evaluated the pair
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Altogether, these data suggest that the upregulation of GSH, rather than the dTTP pool, is responsible for avoiding oncogene-induced replication stress and less susceptibility to JMF4073 in T315I-Bcr-Abl-32D cells
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