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GIP receptor agonism further enhances insulin release and may improve fat metabolism
2 Due to the lack of self-healing capacity of articular cartilage, OA is among the most challenging joint diseases and there is currently no cure for it
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A GPCR negative feedback model recapitulates fine dynamic responses of wt and tax6 mutants The above experiments position calcium and TAX6/Calcineurin as key components for GPCR inhibition and exact adaptation
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