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For instance, manifestation of radiation toxicity at the cellular level involves simultaneous activation of various pro-survival and cytotoxic pathways in a ROS-dependent (indirect effect of radiation) and independent (direct effect of radiation) manner [37, 40, 100]
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Dulaglutide, another GLP-1RA, was found to suppress the proportion of pathogenic Th1 and Th17 cells and inhibit the secretion of granulocyte-macrophage colony-stimulating factor (GM-CSF) by Th1 cells within the CNS of EAE mice, resulting in improved clinical outcomes [268]
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