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Impressive trial results complex realities Despite the promise of incretin-based therapies, achieving similar outcomes outside of clinical trials has been challenging
GSTM2 deletion strongly promotes hepatic fat accumulation To investigate the function of GSTM2 in the progression of hepatic steatosis, we generated a GSTM2 knockout ( GSTM2 KO) mouse (Supplementary Fig
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Disclaimer: Compounded preparations are customized for individual patients and have not been evaluated or approved by the FDA for safety or efficacy
Repeat at week 2 to capture initial IGF-1 and metabolic response
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