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It increases fibroblast migration and dispersal, induces F-actin formation in fibroblasts, modulates angiogenesis, and enhances vascular expression of VEGFR2

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Salidroside ameliorates sepsis-induced acute lung injury and mortality via downregulating NF-B and HMGB1 pathways through the upregulation of SIRT1

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Cancer related mutations in NRF2 impair its recognition by Keap1Cul3 E3 ligase and promote malignancy

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