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5 MQC collapse-induced chondrocyte death: a pivotal event in OA progression With the progression of OA to advanced stages, the compensatory capacity of MQC is thought to become substantially depleted, which may contribute to the breakdown of mitochondrial homeostasis

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These effects were similar in N5-WT and N5-Tr islets, thereby invalidating our earlier observation that the oxidation of cytosolic glutathione upon glucose removal in the presence of H 2 O 2 was slightly lower in BL/6J than BL/6N islets (see Figure 3E in [33])