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Notably, substantial residual cardiovascular risk persists despite therapy with either class alone, reinforcing the rationale for combination therapy, particularly in individuals with established ASCVD, HF, CKD, or overlapping risk domains [11, 16, 17, 38]
No peer-reviewed evidence supports dose adjustments for cosmetic goals
Additionally, COX2 activity in MDSCs is associated with the generation of mitochondrial ROS, and decreasing COX2 reduces ROS levels, compromising MDSCs inhibitory capacity [38, 39]
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Never adjust both compounds simultaneously, as this makes it impossible to identify which compound is responsible for any changes in symptoms or blood markers
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