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Edaravone prevents reactive oxygen species from being formed and/or removes them before they can damage neurons, in effect limiting apopotosis of post synaptic terminals as well as the inactivation of EAAT2 receptors in astrocytes and increasing the rate of glutamate re-uptake (Figure 1)

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Related upstream secretagogues studied in this space include Tesamorelin and Sermorelin

GHK-Cu (glycyl-L-histidyl-L-lysine copper complex) targets hair follicle biology through mechanisms neither of those compounds address: dermal papilla cell proliferation, follicle stem cell recovery, and TGF-beta1 suppression

Czerwinski M et al