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ATM naturally exists as a resting inactive dimer which is auto-phosphorylated into an active monomeric form that interacts with the MRE11/RAD50/NBS1 (MRN) complex at the site of strand breaks, co-ordinating DNA repair 45,46

81 Additionally, quercetin increases the activity of luteinizing hormone, follicle-stimulating hormone, estradiol, and progesterone

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A 2025 study discovered this bacterial enzyme pathway and even identified an inhibitor that could block it [170, 171]

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