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However, a dysregulated increase in the level of these free radicals, as seen in mitochondrial diseases, can result in oxidative damage by overwhelming the cellular antioxidant defence mechanism, of which glutathione is the principal component [4]

Hepatic Detoxification via Glutathione-S-Transferases In hepatocytesthe primary site of xenobiotic metabolismglutathione (GSH) conjugates electrophilic toxins via glutathione-S-transferases (GSTs), enhancing their solubility to facilitate elimination through bile or urine (26)

S.Alzheimer's Disease Neuroimaging Initiative (2024)