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The complementary mechanisms create a multi-phase healing support system: early cytoprotection and inflammation control (all three peptides through distinct pathways), proliferative phase support including angiogenesis (triple-pathway vascular stimulation), cellular coordination, and matrix formation (TB-500 and GHK-Cu), structural integration through periosteal activation and stem cell recruitment (BPC-157), and remodeling phase regulation preventing fibrosis while supporting functional tissue architecture (TB-500's antifibrotic effects and GHK-Cu's MMP regulation)

6,7,8 These proteins have emerged as critical molecular chaperones that facilitate folding, prevent aggregation, and stabilize proteins under stress

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(c) simple administration of peptide in clinical environment

However, bacterial biofilm in chronic wounds may cause irreversible bacterial adhesion, leading to persistent inflammation, tissue damage, and ongoing infection [43, 45, 46]