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Furthermore, they could show that cells lacking both SLC25A39 as well as its paralogue SLC25A40 exhibit defects in the activity and stability of proteins containing ironsulfur clusters (ISCs)
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The study enrolled 1708 patients who were in stable condition, at least 50 years old, and had had a prior heart attack
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doi: 10.1186/s12951-025-03258-5 150 LinYJinJHanQHangDChenZYeSet al
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