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Our experimental research revealed that TNF- significantly inhibits EBV reactivation and that this inhibitory effect is mediated primarily through its receptor TNFR1
In contrast, another study found that late stage of differentiation exposure at 300 nmol/l led to upregulation of lipolytic adipose triglyceride lipase (ATGL), aligning with the known lipolytic effects of GLP-1-RAs in human adipocytes
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