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In our study, we demonstrated a persistent senescent-inflammatory microenvironment and a p53-serine 15 phosphorylation regulatory mechanism in keloids, which may play crucial roles in the aggression and recurrence of keloids
Unlike the Cagri-CTR-Gs complexes, in the CagriBB-bound CTR H296 ECL2 adopted the inward rotamer position that was observed when bound to other non-lipidated peptides, and ECL3 formed the more closed conformation in both experimental repeats
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