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Nevertheless, current evidence supports GLP-1 RAs and multi-agonist formulations as highly promising pharmacotherapies that address the complex, multifactorial pathophysiology of MASLD while simultaneously improving cardiometabolic comorbidities, positioning them as cornerstone treatments in the evolving therapeutic landscape for this prevalent and burdensome disease

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Prednisone raises blood glucose through gluconeogenesis and insulin resistance, while semaglutide lowers glucose by enhancing insulin secretion and slowing gastric emptying

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