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Exosomal miR-4449 derived from DKD patient serum substantially accelerates tubular epithelial cell pyroptosis and oxidative stress through the transcriptional suppression of HIC1, leading to GSDMD-mediated pore formation and inflammatory cytokine secretion (167)

This phase does correspond well to the second phase (first oxidation phase) observed in the 455 nm traces, confirming that reoxidation of the FAD and formation of the thiolate-FAD charge transfer absorption occur concomitantly, establishing that deprotonation of C159 remains rapid

2007 Sep;1(3):201-7