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High levels of MAO-A were found in mitral valve regurgitation, and the MAOs were shown to contribute to the oxidative stress in the heart valve.[ref] These cardiovascular effects highlight that MAO-A is not only a brain enzyme but also a significant source of oxidative stress in the heart and veins
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The mechanistic case for some lean-mass protection during a deep deficit
The type 2 inflammatory response is primarily driven by cytokines like IL-4, IL-5, and IL-13, together with other inflammatory agents produced by Th2 cells and ILC2s [245, 246]
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