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Although these findings of dysbiosis in ME/CFS are evident [12, 21,22,23,24,25,26,27,28,29,30,31], their exact role in the disease mechanism remains unclear [10]

Research Background & Mechanistic Focus Current research use centers on cell-migration, structural-protein, and cellular-response signaling models, comparative presentation or formulation behavior where applicable, and controlled experimental models designed to evaluate compound-specific signaling activity within non-clinical laboratory systems

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