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In summary, the inflammatory response associated with RIRI is characterized by rapid onset, complex regulation, and feedback enhancement, manifesting as a multidimensional pathological mechanism involving TLR/NF-B activation, inflammatory cytokine release, immune cell infiltration, vascular dysfunction, and synergistic activation of the complement system, which together construct a highly dynamic immune-inflammatory network that accelerates renal parenchymal structural damage and functional failure
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114,220,223,224,225 Different CBS inhibitors have been designed and tested over the past years, starting with amino-oxyacetic acid (AOAA) 52,58,60 and related prodrugs
Squid glutathione S-transferase
PMID 27306908
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