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In contrast, SLC25A40 lacks the GSH-sensing cysteines of SLC25A39 and, as independently shown by Birsoys and Shens groups 11,12 , does not undergo post-translational regulation via the GSH-sensing stability loop that controls SLC25A39 protein levels

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However, these mainstream therapies face critical limitations including the rapid emergence of drug resistance, FKS1/FKS2 gene mutations reducing -1,3-glucan synthase affinity, Targeted enzyme (Erg11p) modification, overexpression of efflux pumps (CDR1, MDR1), and upregulation of the ergosterol pathway mediate azole resistance (Pristov and Ghannoum, 2019)

I was a new mom, in a new country, with a new baby, she says

Zhu H, Itoh K, Yamamoto M, Zweier JL, Li Y