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Moreover, mitochondrial dysfunction and related oxidative stress can activate the innate branch of the immune system, leading to the release of pro-inflammatory cytokines influencing various depression-related pathophysiological mechanisms: monoaminergic neurotransmission disruption, tryptophan degradation toward neurotoxic catabolites, glutamate-related excitotoxicity, decreased neurotrophic factors and alterations in the hypothalamic-pituitary-adrenal axis [3, 33, 59]

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doi:10.1016/j.bone.2024.117338 Cheng L, Hu Y, Li YY, et al

Myth: GLP-1 medications destroy testosterone This is the opposite of what the research shows

ACS Biomaterials Science & Engineering