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In murine models of doxorubicin (DOX) and ischemia/reperfusion (I/R)-induced cardiomyopathy, administering DOX to mice induced cardiomyopathy with a rapid, systemic accumulation of non-heme iron via heme degradation by Nrf2-mediated upregulation of HO-1, which effect was abolished in Nrf2-deficient mice (Fang et al., 2019)

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is author on the patent entitled Methods for detecting graft versus host disease, US-61/542, 630

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However, skin hydration, dermal thickness, transepidermal water loss (TEWL), and viscoelasticity were not significantly altered [34]