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In line with the idea that peripheral immune cells are not passive responders but function as key hubs linking systemic inflammation to central neurodegeneration, we propose that peripheral effects should be considered as mechanistic contributors to neuronal death in PD, as clinically accessible targets for biomarker development, and as potential therapeutic strategies to slow and hopefully halt its progression

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Adrenergic signaling in muscularis macrophages limits infection-induced neuronal loss

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