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Furthermore, GLP-1 receptor agonists protect against neurodegeneration by suppressing tau hyperphosphorylation
In contrast, direct central mechanisms including PI3K/Akt-mediated neuroprotection, BDNF upregulation, and enhanced autophagy are primarily supported by preclinical data obtained at drug concentrations substantially higher than those achievable in the human brain at therapeutic doses
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A 2025 systematic review (PMC, 36 studies, 1993-2024) found consistent improvement in tendon, ligament, muscle, and bone healing in animal models
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