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The copper interaction anomalies in these proteins contribute to misfolding and aggregation that is linked to neurodegeneration

No saturation behavior for GSSG was previously detected for the fusion construct between rxYFP and ScGrx1 31 , which makes sense considering that PfGrx and many other glutaredoxins are at least two orders of magnitude more active than ScGrx1 (Table 1) 14,56

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