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(2024) employed a maternal immune activation (MIA) mouse model, induced by lipopolysaccharides (LPS) administration during pregnancy, which led to autism-like behaviors in the offspring
The published rationale is that an amylin-receptor agonist and a GLP-1 receptor agonist engage distinct but complementary signaling systems
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[unverified no linkable source] UK laboratory quality and MHRA medicinal-products framework context (ICH Q7 / ISO 9001 alignment)
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