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GLP-1 activates the GLP-1 receptor (GLP-1R) in a glucose-dependent manner to potentiate insulin release and suppress glucagon secretion, and thereby lead to blood sugar lowering and weight loss [3][4]
Caloric restriction during weight loss can temporarily affect thyroid hormone levels , which may contribute to cold sensitivity without representing true hypothyroidism
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The arrival of oral GLP-1 therapies marks the beginning of a new chapter in obesity pharmacotherapy.
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Peak appetite suppression typically occurs 24 to 72 hours after injection
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